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Passive Protective Effects of Chicken Egg Yolk Immunoglobulins ( IgY ) Against Anguillid Herpesvirus Infection in American Eel ( Anguilla rostrata )

Ying‐Ying Li; Qiang Chen; Wen‐Zhong Chen; Jin‐Xian Yang; Jun‐Qing Ge
Journal of Fish Diseases · 2026

Abstract

Anguillid herpesvirus (AngHV) is the causative agent of ‘mucus sloughing and hemorrhagic septicemia disease’ in eels. Egg yolk immunoglobulin (IgY) has been reported to serve as oral administration of antibodies against bacterial and viral pathogens. In this study, an inactivated vaccine of AngHV was developed to immunise the laying hens, thereby facilitating the production of anti‐AngHV IgY. The yolks were subsequently collected and processed into a dry yolk powder, and IgY was extracted from the yolk powder. The anti‐AngHV efficacy of the IgY was assessed through both in vitro and in vivo assays. In vitro analyses showed that the purified IgY was capable of neutralizing AngHV in an antibody concentration‐dependent manner. In the in vivo experiment, the incorporation of 6‰ yolk powder into the diet of eels exhibited no negative impact on growth performance, intestinal tissue morphology, intestinal microbiota, and metabolic profiles. However, the incidence of morbidity in AngHV‐infected eels decreased from 100% to 71%, while mortality rates declined from 57.14% to 28.57%. Additionally, the concentration of AngHV in the skin mucus was significantly reduced. This study provides the evidence that the anti‐AngHV IgY can effectively confer protection to eels against AngHV infection, thereby establishing a foundation for the development of anti‐AngHV therapy products using the yolk powder.

Bibliographic Information

JournalJournal of Fish Diseases
PublisherWiley
Publication Date2026-07-02
Publication Year2026
Document TypeJournal Article
Print ISSN0140-7775
eISSN1365-2761
DOI10.1111/jfd.70230
SubjectGeneral Aquaculture, Fisheries & Fish Science

Access Information

NARA Access Coverage1997-01-01~Current
Journal Homepagehttps://onlinelibrary.wiley.com/loi/13652761
Publisher PageOpen Publisher Page
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