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Springer Nature3
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Pflügers Archiv - European Journal of Physiology3
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Benjamin Soret results 3 · Newest (Page 1/1, per page 25)
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K2P2.1 channels modulate the pH- and mechanosensitivity of pancreatic stellate cellsNARA Subscribed
Pflügers Archiv - European Journal of Physiology · 2025 · Vol. 477 · Issue 1 · Springer
Micol Rugi; Verena Hofschröer; Zoltán Pethő; Benjamin Soret; Thorsten Loeck; Albrecht Schwab
Pancreatic stellate cells (PSCs) are central in the development of acute pancreatitis and tumor fibrosis in pancreatic ductal adenocarcinoma (PDAC). Fibrosis and a unique pH landscape represent characteristic properties of the PDAC microenvironment. Mechanosensitive ion channels are involved in the activation of PSCs. Among these channels, K 2P 2.1 has not yet been studied in PSCs. K 2P 2.1 channels are pH- and mechanosensitiv...
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Correction to: The context‑dependent role of the Na+/Ca2+‑exchanger (NCX) in pancreatic stellate cell migrationNARA Subscribed
Pflügers Archiv - European Journal of Physiology · 2024 · Vol. 476 · Issue 1 · Springer
Thorsten Loeck; Micol Rugi; Luca Matteo Todesca; Paulina Kalinowska; Benjamin Soret; Ilka Neumann; Sandra Schimmelpfennig; Karolina Najder; Zoltán Pethő; Valerio Farfariello; Natalia Prevarskaya; Albrecht Schwab
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The context-dependent role of the Na+/Ca2+-exchanger (NCX) in pancreatic stellate cell migrationNARA Subscribed
Pflügers Archiv - European Journal of Physiology · 2023 · Vol. 475 · Issue 10 · Springer
Thorsten Loeck; Micol Rugi; Luca Matteo Todesca; Paulina Kalinowska; Benjamin Soret; Ilka Neumann; Sandra Schimmelpfennig; Karolina Najder; Zoltán Pethő; Valerio Farfariello; Natalia Prevarskaya; Albrecht Schwab
Pancreatic stellate cells (PSCs) that can co-metastasize with cancer cells shape the tumor microenvironment (TME) in pancreatic ductal adenocarcinoma (PDAC) by producing an excessive amount of extracellular matrix. This leads to a TME characterized by increased tissue pressure, hypoxia, and acidity. Moreover, cells within the tumor secrete growth factors. The stimuli of the TME trigger Ca 2+ signaling and cellular Na + loading...
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